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anti-p62/ SQSTM1 (C-terminus) guinea pig polyclonal, serum

Rat colon (courtesy of J. Hess, University Hospital Heidelberg)

WB with anti-p62 antibody (Cat. No. GP62-C, 1:1000), PLC whole cell lysate (2.5 - 10 ug)

Human PLC cells

WB with anti-p62 antibody (Cat. No. GP62-C, 1:1000) on different human cell lines

Human kidney (courtesy of J. Hess, University Hospital Heidelberg)

Human colon (courtesy of J. Hess, University Hospital Heidelberg)

Rat skeletal muscle (courtesy of J. Hess, University Hospital Heidelberg)

Mouse skeletal muscle (courtesy of J. Hess, University Hospital Heidelberg)

Mouse stomach (courtesy of J. Hess, University Hospital Heidelberg)

Mouse colon (courtesy of J. Hess, University Hospital Heidelberg)

Rat stomach (courtesy of J. Hess, University Hospital Heidelberg)

Rat colon (courtesy of J. Hess, University Hospital Heidelberg)

Loss of ALS2/Alsin exacerbates motor dysfunction in a SOD1-expressing mouse ALS model by disturbing endolysosomal trafficking.

Hadano, S., Otomo, A., et al. Loss of ALS2/Alsin exacerbates motor dysfunction in a SOD1-expressing mouse ALS model by disturbing endolysosomal trafficking. PLoS One. 2010-03-27.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of ALS2/Alsin exacerbates motor dysfunction in a SOD1-expressing mouse ALS model by disturbing endolysosomal trafficking.

Hadano, S., Otomo, A., et al. Loss of ALS2/Alsin exacerbates motor dysfunction in a SOD1-expressing mouse ALS model by disturbing endolysosomal trafficking. PLoS One. 2010-03-27.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Inhibition of mTOR by rapamycin abolishes cognitive deficits and reduces amyloid-beta levels in a mouse model of Alzheimer's disease.

Spilman, P., Podlutskaya, N., et al. Inhibition of mTOR by rapamycin abolishes cognitive deficits and reduces amyloid-beta levels in a mouse model of Alzheimer's disease. PLoS One. 2010-04-09.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Targeting protective autophagy exacerbates UV-triggered apoptotic cell death.

Chen, L. H., Chu, P. M., et al. Targeting protective autophagy exacerbates UV-triggered apoptotic cell death. Int J Mol Sci. 2012-02-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cell loss and autophagy in the extra-adrenal chromaffin organ of Zuckerkandl are regulated by glucocorticoid signalling.

Schober, A., Parlato, R., et al. Cell loss and autophagy in the extra-adrenal chromaffin organ of Zuckerkandl are regulated by glucocorticoid signalling. J Neuroendocrinol. 2013-01-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cell loss and autophagy in the extra-adrenal chromaffin organ of Zuckerkandl are regulated by glucocorticoid signalling.

Schober, A., Parlato, R., et al. Cell loss and autophagy in the extra-adrenal chromaffin organ of Zuckerkandl are regulated by glucocorticoid signalling. J Neuroendocrinol. 2013-01-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Rapamycin attenuates the progression of tau pathology in P301S tau transgenic mice.

Ozcelik, S., Fraser, G., et al. Rapamycin attenuates the progression of tau pathology in P301S tau transgenic mice. PLoS One. 2013-05-15.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

A novel acylaminoimidazole derivative, WN1316, alleviates disease progression via suppression of glial inflammation in ALS mouse model.

Tanaka, K., Kanno, T., et al. A novel acylaminoimidazole derivative, WN1316, alleviates disease progression via suppression of glial inflammation in ALS mouse model. PLoS One. 2014-02-06.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

A novel acylaminoimidazole derivative, WN1316, alleviates disease progression via suppression of glial inflammation in ALS mouse model.

Tanaka, K., Kanno, T., et al. A novel acylaminoimidazole derivative, WN1316, alleviates disease progression via suppression of glial inflammation in ALS mouse model. PLoS One. 2014-02-06.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Rotavirus increases levels of lipidated LC3 supporting accumulation of infectious progeny virus without inducing autophagosome formation.

Arnoldi, F., De Lorenzo, G., et al. Rotavirus increases levels of lipidated LC3 supporting accumulation of infectious progeny virus without inducing autophagosome formation. PLoS One. 2014-04-17.

Species/Reactant: Chlorocebus aethiops (Grivet monkey)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Rotavirus increases levels of lipidated LC3 supporting accumulation of infectious progeny virus without inducing autophagosome formation.

Arnoldi, F., De Lorenzo, G., et al. Rotavirus increases levels of lipidated LC3 supporting accumulation of infectious progeny virus without inducing autophagosome formation. PLoS One. 2014-04-17.

Species/Reactant: Chlorocebus aethiops (Grivet monkey)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Ribosomal protein mutations induce autophagy through S6 kinase inhibition of the insulin pathway.

Heijnen, H. F., van Wijk, R., et al. Ribosomal protein mutations induce autophagy through S6 kinase inhibition of the insulin pathway. PLoS Genet. 2014-05-31.

Species/Reactant: Danio rerio (Zebrafish)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Ribosomal protein mutations induce autophagy through S6 kinase inhibition of the insulin pathway.

Heijnen, H. F., van Wijk, R., et al. Ribosomal protein mutations induce autophagy through S6 kinase inhibition of the insulin pathway. PLoS Genet. 2014-05-31.

Species/Reactant: Homo sapiens (Human)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Ribosomal protein mutations induce autophagy through S6 kinase inhibition of the insulin pathway.

Heijnen, H. F., van Wijk, R., et al. Ribosomal protein mutations induce autophagy through S6 kinase inhibition of the insulin pathway. PLoS Genet. 2014-05-31.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Ribosomal protein mutations induce autophagy through S6 kinase inhibition of the insulin pathway.

Heijnen, H. F., van Wijk, R., et al. Ribosomal protein mutations induce autophagy through S6 kinase inhibition of the insulin pathway. PLoS Genet. 2014-05-31.

Species/Reactant: Danio rerio (Zebrafish)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death.

Liu, S., Sarkar, C., et al. Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death. Cell Death Dis. 2015-01-08.

Species/Reactant: Rattus norvegicus (Rat)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death.

Liu, S., Sarkar, C., et al. Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death. Cell Death Dis. 2015-01-08.

Species/Reactant: Rattus norvegicus (Rat)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death.

Liu, S., Sarkar, C., et al. Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death. Cell Death Dis. 2015-01-08.

Species/Reactant: Rattus norvegicus (Rat)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death.

Liu, S., Sarkar, C., et al. Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death. Cell Death Dis. 2015-01-08.

Species/Reactant: Rattus norvegicus (Rat)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death.

Liu, S., Sarkar, C., et al. Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death. Cell Death Dis. 2015-01-08.

Species/Reactant: Rattus norvegicus (Rat)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death.

Liu, S., Sarkar, C., et al. Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death. Cell Death Dis. 2015-01-08.

Species/Reactant: Rattus norvegicus (Rat)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cucurbitacin E has neuroprotective properties and autophagic modulating activities on dopaminergic neurons.

Arel-Dubeau, A. M., Longpré, F., et al. Cucurbitacin E has neuroprotective properties and autophagic modulating activities on dopaminergic neurons. Oxid Med Cell Longev. 2015-01-13.

Species/Reactant: Rattus norvegicus (Rat)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates.

Morimoto, D., Walinda, E., et al. The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates. Nat Commun. 2015-01-20.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates.

Morimoto, D., Walinda, E., et al. The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates. Nat Commun. 2015-01-20.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates.

Morimoto, D., Walinda, E., et al. The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates. Nat Commun. 2015-01-20.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates.

Morimoto, D., Walinda, E., et al. The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates. Nat Commun. 2015-01-20.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates.

Morimoto, D., Walinda, E., et al. The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates. Nat Commun. 2015-01-20.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Lim, J., Lachenmayer, M. L., et al. Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates. PLoS Genet. 2015-02-28.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Lim, J., Lachenmayer, M. L., et al. Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates. PLoS Genet. 2015-02-28.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Lim, J., Lachenmayer, M. L., et al. Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates. PLoS Genet. 2015-02-28.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Lim, J., Lachenmayer, M. L., et al. Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates. PLoS Genet. 2015-02-28.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Lim, J., Lachenmayer, M. L., et al. Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates. PLoS Genet. 2015-02-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Lim, J., Lachenmayer, M. L., et al. Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates. PLoS Genet. 2015-02-28.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Lim, J., Lachenmayer, M. L., et al. Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates. PLoS Genet. 2015-02-28.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Lim, J., Lachenmayer, M. L., et al. Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates. PLoS Genet. 2015-02-28.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Lim, J., Lachenmayer, M. L., et al. Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates. PLoS Genet. 2015-02-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Lim, J., Lachenmayer, M. L., et al. Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates. PLoS Genet. 2015-02-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Lim, J., Lachenmayer, M. L., et al. Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates. PLoS Genet. 2015-02-28.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagic signaling and proteolytic enzyme activity in cardiac and skeletal muscle of spontaneously hypertensive rats following chronic aerobic exercise.

McMillan, E. M., Paré, M. F., et al. Autophagic signaling and proteolytic enzyme activity in cardiac and skeletal muscle of spontaneously hypertensive rats following chronic aerobic exercise. PLoS One. 2015-03-24.

Species/Reactant: Rattus norvegicus (Rat)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Estrogen receptor α regulates non-canonical autophagy that provides stress resistance to neuroblastoma and breast cancer cells and involves BAG3 function.

Felzen, V., Hiebel, C., et al. Estrogen receptor α regulates non-canonical autophagy that provides stress resistance to neuroblastoma and breast cancer cells and involves BAG3 function. Cell Death Dis. 2015-07-09.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Frontotemporal dementia caused by CHMP2B mutation is characterised by neuronal lysosomal storage pathology.

Clayton, E. L., Mizielinska, S., et al. Frontotemporal dementia caused by CHMP2B mutation is characterised by neuronal lysosomal storage pathology. Acta Neuropathol. 2015-10-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Frontotemporal dementia caused by CHMP2B mutation is characterised by neuronal lysosomal storage pathology.

Clayton, E. L., Mizielinska, S., et al. Frontotemporal dementia caused by CHMP2B mutation is characterised by neuronal lysosomal storage pathology. Acta Neuropathol. 2015-10-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Frontotemporal dementia caused by CHMP2B mutation is characterised by neuronal lysosomal storage pathology.

Clayton, E. L., Mizielinska, S., et al. Frontotemporal dementia caused by CHMP2B mutation is characterised by neuronal lysosomal storage pathology. Acta Neuropathol. 2015-10-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Frontotemporal dementia caused by CHMP2B mutation is characterised by neuronal lysosomal storage pathology.

Clayton, E. L., Mizielinska, S., et al. Frontotemporal dementia caused by CHMP2B mutation is characterised by neuronal lysosomal storage pathology. Acta Neuropathol. 2015-10-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

All-trans retinoic acid and rapamycin normalize Hutchinson Gilford progeria fibroblast phenotype.

Pellegrini, C., Columbaro, M., et al. All-trans retinoic acid and rapamycin normalize Hutchinson Gilford progeria fibroblast phenotype. Oncotarget. 2015-10-06.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Sqstm1-GFP knock-in mice reveal dynamic actions of Sqstm1 during autophagy and under stress conditions in living cells.

Eino, A., Kageyama, S., et al. Sqstm1-GFP knock-in mice reveal dynamic actions of Sqstm1 during autophagy and under stress conditions in living cells. J Cell Sci. 2015-12-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Sqstm1-GFP knock-in mice reveal dynamic actions of Sqstm1 during autophagy and under stress conditions in living cells.

Eino, A., Kageyama, S., et al. Sqstm1-GFP knock-in mice reveal dynamic actions of Sqstm1 during autophagy and under stress conditions in living cells. J Cell Sci. 2015-12-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Sqstm1-GFP knock-in mice reveal dynamic actions of Sqstm1 during autophagy and under stress conditions in living cells.

Eino, A., Kageyama, S., et al. Sqstm1-GFP knock-in mice reveal dynamic actions of Sqstm1 during autophagy and under stress conditions in living cells. J Cell Sci. 2015-12-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Sqstm1-GFP knock-in mice reveal dynamic actions of Sqstm1 during autophagy and under stress conditions in living cells.

Eino, A., Kageyama, S., et al. Sqstm1-GFP knock-in mice reveal dynamic actions of Sqstm1 during autophagy and under stress conditions in living cells. J Cell Sci. 2015-12-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy and endosomal trafficking inhibition by Vibrio cholerae MARTX toxin phosphatidylinositol-3-phosphate-specific phospholipase A1 activity.

Agarwal, S., Kim, H., et al. Autophagy and endosomal trafficking inhibition by Vibrio cholerae MARTX toxin phosphatidylinositol-3-phosphate-specific phospholipase A1 activity. Nat Commun. 2015-10-26.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Puvirajesinghe, T. M., Bertucci, F., et al. Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer. Nat Commun. 2016-01-12.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Puvirajesinghe, T. M., Bertucci, F., et al. Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer. Nat Commun. 2016-01-12.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Puvirajesinghe, T. M., Bertucci, F., et al. Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer. Nat Commun. 2016-01-12.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Puvirajesinghe, T. M., Bertucci, F., et al. Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer. Nat Commun. 2016-01-12.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Puvirajesinghe, T. M., Bertucci, F., et al. Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer. Nat Commun. 2016-01-12.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Puvirajesinghe, T. M., Bertucci, F., et al. Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer. Nat Commun. 2016-01-12.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Puvirajesinghe, T. M., Bertucci, F., et al. Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer. Nat Commun. 2016-01-12.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Puvirajesinghe, T. M., Bertucci, F., et al. Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer. Nat Commun. 2016-01-12.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Puvirajesinghe, T. M., Bertucci, F., et al. Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer. Nat Commun. 2016-01-12.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy.

Taneike, M., Nishida, K., et al. mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy. PLoS One. 2016-03-31.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy.

Taneike, M., Nishida, K., et al. mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy. PLoS One. 2016-03-31.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy.

Taneike, M., Nishida, K., et al. mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy. PLoS One. 2016-03-31.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy.

Taneike, M., Nishida, K., et al. mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy. PLoS One. 2016-03-31.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy.

Taneike, M., Nishida, K., et al. mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy. PLoS One. 2016-03-31.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy.

Taneike, M., Nishida, K., et al. mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy. PLoS One. 2016-03-31.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

A Founder Mutation in VPS11 Causes an Autosomal Recessive Leukoencephalopathy Linked to Autophagic Defects.

Zhang, J., Lachance, V., et al. A Founder Mutation in VPS11 Causes an Autosomal Recessive Leukoencephalopathy Linked to Autophagic Defects. PLoS Genet. 2016-04-01.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming.

Saito, T., Ichimura, Y., et al. p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming. Nat Commun. 2016-06-27.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming.

Saito, T., Ichimura, Y., et al. p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming. Nat Commun. 2016-06-27.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming.

Saito, T., Ichimura, Y., et al. p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming. Nat Commun. 2016-06-27.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming.

Saito, T., Ichimura, Y., et al. p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming. Nat Commun. 2016-06-27.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming.

Saito, T., Ichimura, Y., et al. p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming. Nat Commun. 2016-06-27.

Species/Reactant: Homo sapiens (Human)
Applications: Immunohistochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming.

Saito, T., Ichimura, Y., et al. p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming. Nat Commun. 2016-06-27.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

TSC but not PTEN loss in starving cones of retinitis pigmentosa mice leads to an autophagy defect and mTORC1 dissociation from the lysosome.

Venkatesh, A., Ma, S., et al. TSC but not PTEN loss in starving cones of retinitis pigmentosa mice leads to an autophagy defect and mTORC1 dissociation from the lysosome. Cell Death Dis. 2016-06-30.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

TSC but not PTEN loss in starving cones of retinitis pigmentosa mice leads to an autophagy defect and mTORC1 dissociation from the lysosome.

Venkatesh, A., Ma, S., et al. TSC but not PTEN loss in starving cones of retinitis pigmentosa mice leads to an autophagy defect and mTORC1 dissociation from the lysosome. Cell Death Dis. 2016-06-30.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

TSC but not PTEN loss in starving cones of retinitis pigmentosa mice leads to an autophagy defect and mTORC1 dissociation from the lysosome.

Venkatesh, A., Ma, S., et al. TSC but not PTEN loss in starving cones of retinitis pigmentosa mice leads to an autophagy defect and mTORC1 dissociation from the lysosome. Cell Death Dis. 2016-06-30.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

TSC but not PTEN loss in starving cones of retinitis pigmentosa mice leads to an autophagy defect and mTORC1 dissociation from the lysosome.

Venkatesh, A., Ma, S., et al. TSC but not PTEN loss in starving cones of retinitis pigmentosa mice leads to an autophagy defect and mTORC1 dissociation from the lysosome. Cell Death Dis. 2016-06-30.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Intracellular glutathione determines bortezomib cytotoxicity in multiple myeloma cells.

Starheim, K. K., Holien, T., et al. Intracellular glutathione determines bortezomib cytotoxicity in multiple myeloma cells. Blood Cancer J. 2016-07-15.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Effect of Exendin-4 on Autophagy Clearance in Beta Cell of Rats with Tacrolimus-induced Diabetes Mellitus.

Lim, S. W., Jin, L., et al. Effect of Exendin-4 on Autophagy Clearance in Beta Cell of Rats with Tacrolimus-induced Diabetes Mellitus. Sci Rep. 2016-07-20.

Species/Reactant: Rattus norvegicus (Rat)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Oleuropein Prevents Neuronal Death, Mitigates Mitochondrial Superoxide Production and Modulates Autophagy in a Dopaminergic Cellular Model.

Achour, I., Arel-Dubeau, A. M., et al. Oleuropein Prevents Neuronal Death, Mitigates Mitochondrial Superoxide Production and Modulates Autophagy in a Dopaminergic Cellular Model. Int J Mol Sci. 2016-08-09.

Species/Reactant: Rattus norvegicus (Rat)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Hydroxychloroquine potentiates carfilzomib toxicity towards myeloma cells.

Baranowska, K., Misund, K., et al. Hydroxychloroquine potentiates carfilzomib toxicity towards myeloma cells. Oncotarget. 2016-10-25.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Hydroxychloroquine potentiates carfilzomib toxicity towards myeloma cells.

Baranowska, K., Misund, K., et al. Hydroxychloroquine potentiates carfilzomib toxicity towards myeloma cells. Oncotarget. 2016-10-25.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Differential roles of NF-Y transcription factor in ER chaperone expression and neuronal maintenance in the CNS.

Yamanaka, T., Tosaki, A., et al. Differential roles of NF-Y transcription factor in ER chaperone expression and neuronal maintenance in the CNS. Sci Rep. 2016-09-30.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Differential roles of NF-Y transcription factor in ER chaperone expression and neuronal maintenance in the CNS.

Yamanaka, T., Tosaki, A., et al. Differential roles of NF-Y transcription factor in ER chaperone expression and neuronal maintenance in the CNS. Sci Rep. 2016-09-30.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Differential roles of NF-Y transcription factor in ER chaperone expression and neuronal maintenance in the CNS.

Yamanaka, T., Tosaki, A., et al. Differential roles of NF-Y transcription factor in ER chaperone expression and neuronal maintenance in the CNS. Sci Rep. 2016-09-30.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Activation of the Keap1/Nrf2 stress response pathway in autophagic vacuolar myopathies.

Duleh, S., Wang, X., et al. Activation of the Keap1/Nrf2 stress response pathway in autophagic vacuolar myopathies. Acta Neuropathol Commun. 2016-10-31.

Species/Reactant: Homo sapiens (Human)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Activation of the Keap1/Nrf2 stress response pathway in autophagic vacuolar myopathies.

Duleh, S., Wang, X., et al. Activation of the Keap1/Nrf2 stress response pathway in autophagic vacuolar myopathies. Acta Neuropathol Commun. 2016-10-31.

Species/Reactant: Homo sapiens (Human)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Activation of the Keap1/Nrf2 stress response pathway in autophagic vacuolar myopathies.

Duleh, S., Wang, X., et al. Activation of the Keap1/Nrf2 stress response pathway in autophagic vacuolar myopathies. Acta Neuropathol Commun. 2016-10-31.

Species/Reactant: Homo sapiens (Human)
Applications: Immunohistochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Activation of the Keap1/Nrf2 stress response pathway in autophagic vacuolar myopathies.

Duleh, S., Wang, X., et al. Activation of the Keap1/Nrf2 stress response pathway in autophagic vacuolar myopathies. Acta Neuropathol Commun. 2016-10-31.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy dysregulation in Danon disease.

Nascimbeni, A. C., Fanin, M., et al. Autophagy dysregulation in Danon disease. Cell Death Dis. 2017-01-19.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy dysregulation in Danon disease.

Nascimbeni, A. C., Fanin, M., et al. Autophagy dysregulation in Danon disease. Cell Death Dis. 2017-01-19.

Species/Reactant: Homo sapiens (Human)
Applications: Immunohistochemistry-frozen immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy dysregulation in Danon disease.

Nascimbeni, A. C., Fanin, M., et al. Autophagy dysregulation in Danon disease. Cell Death Dis. 2017-01-19.

Species/Reactant: Homo sapiens (Human)
Applications: Immunohistochemistry-frozen immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Gastrin activates autophagy and increases migration and survival of gastric adenocarcinoma cells.

Rao, S. V., Solum, G., et al. Gastrin activates autophagy and increases migration and survival of gastric adenocarcinoma cells. BMC Cancer. 2017-01-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Gastrin activates autophagy and increases migration and survival of gastric adenocarcinoma cells.

Rao, S. V., Solum, G., et al. Gastrin activates autophagy and increases migration and survival of gastric adenocarcinoma cells. BMC Cancer. 2017-01-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Gastrin activates autophagy and increases migration and survival of gastric adenocarcinoma cells.

Rao, S. V., Solum, G., et al. Gastrin activates autophagy and increases migration and survival of gastric adenocarcinoma cells. BMC Cancer. 2017-01-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Gastrin activates autophagy and increases migration and survival of gastric adenocarcinoma cells.

Rao, S. V., Solum, G., et al. Gastrin activates autophagy and increases migration and survival of gastric adenocarcinoma cells. BMC Cancer. 2017-01-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Propagation of pathological α-synuclein in marmoset brain.

Shimozawa, A., Ono, M., et al. Propagation of pathological α-synuclein in marmoset brain. Acta Neuropathol Commun. 2017-02-02.

Species/Reactant: Callithrix jacchus (Common marmoset)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Sarcolipin deletion exacerbates soleus muscle atrophy and weakness in phospholamban overexpressing mice.

Fajardo, V. A., Gamu, D., et al. Sarcolipin deletion exacerbates soleus muscle atrophy and weakness in phospholamban overexpressing mice. PLoS One. 2017-03-10.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Sergin, I., Evans, T. D., et al. Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis. Nat Commun. 2017-06-07.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Sergin, I., Evans, T. D., et al. Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis. Nat Commun. 2017-06-07.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Sergin, I., Evans, T. D., et al. Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis. Nat Commun. 2017-06-07.

Species/Reactant: Mus musculus (House mouse)
Applications: Proximity Ligation Assay

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Sergin, I., Evans, T. D., et al. Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis. Nat Commun. 2017-06-07.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Sergin, I., Evans, T. D., et al. Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis. Nat Commun. 2017-06-07.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Sergin, I., Evans, T. D., et al. Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis. Nat Commun. 2017-06-07.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Sergin, I., Evans, T. D., et al. Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis. Nat Commun. 2017-06-07.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Sergin, I., Evans, T. D., et al. Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis. Nat Commun. 2017-06-07.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Impaired autophagosome clearance contributes to neuronal death in a piglet model of neonatal hypoxic-ischemic encephalopathy.

Cui, D., Sun, D., et al. Impaired autophagosome clearance contributes to neuronal death in a piglet model of neonatal hypoxic-ischemic encephalopathy. Cell Death Dis. 2017-07-13.

Species/Reactant: Sus scrofa domesticus (Domestic pig)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cryptic amyloidogenic elements in mutant NEFH causing Charcot-Marie-Tooth 2 trigger aggresome formation and neuronal death.

Jacquier, A., Delorme, C., et al. Cryptic amyloidogenic elements in mutant NEFH causing Charcot-Marie-Tooth 2 trigger aggresome formation and neuronal death. Acta Neuropathol Commun. 2017-07-14.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cryptic amyloidogenic elements in mutant NEFH causing Charcot-Marie-Tooth 2 trigger aggresome formation and neuronal death.

Jacquier, A., Delorme, C., et al. Cryptic amyloidogenic elements in mutant NEFH causing Charcot-Marie-Tooth 2 trigger aggresome formation and neuronal death. Acta Neuropathol Commun. 2017-07-14.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cryptic amyloidogenic elements in mutant NEFH causing Charcot-Marie-Tooth 2 trigger aggresome formation and neuronal death.

Jacquier, A., Delorme, C., et al. Cryptic amyloidogenic elements in mutant NEFH causing Charcot-Marie-Tooth 2 trigger aggresome formation and neuronal death. Acta Neuropathol Commun. 2017-07-14.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Joachim, J., Razi, M., et al. Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy. Curr Biol. 2017-07-24.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Joachim, J., Razi, M., et al. Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy. Curr Biol. 2017-07-24.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Joachim, J., Razi, M., et al. Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy. Curr Biol. 2017-07-24.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Joachim, J., Razi, M., et al. Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy. Curr Biol. 2017-07-24.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Joachim, J., Razi, M., et al. Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy. Curr Biol. 2017-07-24.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Joachim, J., Razi, M., et al. Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy. Curr Biol. 2017-07-24.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Joachim, J., Razi, M., et al. Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy. Curr Biol. 2017-07-24.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Hypoxia ameliorates intestinal inflammation through NLRP3/mTOR downregulation and autophagy activation.

Cosín-Roger, J., Simmen, S., et al. Hypoxia ameliorates intestinal inflammation through NLRP3/mTOR downregulation and autophagy activation. Nat Commun. 2017-07-24.

Species/Reactant: Homo sapiens (Human)
Applications: Immunohistochemistry-paraffin

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Lysosomotropism depends on glucose: a chloroquine resistance mechanism.

Gallagher, L. E., Radhi, O. A., et al. Lysosomotropism depends on glucose: a chloroquine resistance mechanism. Cell Death Dis. 2017-08-24.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells.

Rivera Vargas, T., Cai, Z., et al. Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells. Nat Commun. 2017-09-15.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells.

Rivera Vargas, T., Cai, Z., et al. Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells. Nat Commun. 2017-09-15.

Species/Reactant: Mus musculus (House mouse)
Applications: Proximity Ligation Assay

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells.

Rivera Vargas, T., Cai, Z., et al. Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells. Nat Commun. 2017-09-15.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells.

Rivera Vargas, T., Cai, Z., et al. Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells. Nat Commun. 2017-09-15.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells.

Rivera Vargas, T., Cai, Z., et al. Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells. Nat Commun. 2017-09-15.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells.

Rivera Vargas, T., Cai, Z., et al. Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells. Nat Commun. 2017-09-15.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Standard Assays for the Study of Autophagy in the Ex Vivo Retina.

Gómez-Sintes, R., Villarejo-Zori, B., et al. Standard Assays for the Study of Autophagy in the Ex Vivo Retina. Cells. 2017-10-22.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Phosphorylation and degradation of αB-crystallin during enterovirus infection facilitates viral replication and induces viral pathogenesis.

Fung, G., Wong, J., et al. Phosphorylation and degradation of αB-crystallin during enterovirus infection facilitates viral replication and induces viral pathogenesis. Oncotarget. 2017-09-26.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

L-leucine and SPNS1 coordinately ameliorate dysfunction of autophagy in mouse and human Niemann-Pick type C disease.

Yanagisawa, H., Ishii, T., et al. L-leucine and SPNS1 coordinately ameliorate dysfunction of autophagy in mouse and human Niemann-Pick type C disease. Sci Rep. 2017-11-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

L-leucine and SPNS1 coordinately ameliorate dysfunction of autophagy in mouse and human Niemann-Pick type C disease.

Yanagisawa, H., Ishii, T., et al. L-leucine and SPNS1 coordinately ameliorate dysfunction of autophagy in mouse and human Niemann-Pick type C disease. Sci Rep. 2017-11-21.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Sequestosome 1 Deficiency Delays, but Does Not Prevent Brain Damage Formation Following Acute Brain Injury in Adult Mice.

Sebastiani, A., Gölz, C., et al. Sequestosome 1 Deficiency Delays, but Does Not Prevent Brain Damage Formation Following Acute Brain Injury in Adult Mice. Front Neurosci. 2018-01-10.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Sequestosome 1 Deficiency Delays, but Does Not Prevent Brain Damage Formation Following Acute Brain Injury in Adult Mice.

Sebastiani, A., Gölz, C., et al. Sequestosome 1 Deficiency Delays, but Does Not Prevent Brain Damage Formation Following Acute Brain Injury in Adult Mice. Front Neurosci. 2018-01-10.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Sequestosome 1 Deficiency Delays, but Does Not Prevent Brain Damage Formation Following Acute Brain Injury in Adult Mice.

Sebastiani, A., Gölz, C., et al. Sequestosome 1 Deficiency Delays, but Does Not Prevent Brain Damage Formation Following Acute Brain Injury in Adult Mice. Front Neurosci. 2018-01-10.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Kageyama, S., Saito, T., et al. Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant. Mol Cell Biol. 2018-04-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Kageyama, S., Saito, T., et al. Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant. Mol Cell Biol. 2018-04-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Kageyama, S., Saito, T., et al. Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant. Mol Cell Biol. 2018-04-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Kageyama, S., Saito, T., et al. Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant. Mol Cell Biol. 2018-04-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Kageyama, S., Saito, T., et al. Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant. Mol Cell Biol. 2018-04-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Kageyama, S., Saito, T., et al. Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant. Mol Cell Biol. 2018-04-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Kageyama, S., Saito, T., et al. Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant. Mol Cell Biol. 2018-04-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Kageyama, S., Saito, T., et al. Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant. Mol Cell Biol. 2018-04-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Kageyama, S., Saito, T., et al. Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant. Mol Cell Biol. 2018-04-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Kageyama, S., Saito, T., et al. Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant. Mol Cell Biol. 2018-04-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Carroll, B., Otten, E. G., et al. Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis. Nat Commun. 2018-01-17.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Carroll, B., Otten, E. G., et al. Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis. Nat Commun. 2018-01-17.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Carroll, B., Otten, E. G., et al. Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis. Nat Commun. 2018-01-17.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Carroll, B., Otten, E. G., et al. Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis. Nat Commun. 2018-01-17.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Carroll, B., Otten, E. G., et al. Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis. Nat Commun. 2018-01-17.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Carroll, B., Otten, E. G., et al. Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis. Nat Commun. 2018-01-17.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Carroll, B., Otten, E. G., et al. Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis. Nat Commun. 2018-01-17.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Sato, S., Uchihara, T., et al. Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice. Sci Rep. 2018-02-12.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Sato, S., Uchihara, T., et al. Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice. Sci Rep. 2018-02-12.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Sato, S., Uchihara, T., et al. Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice. Sci Rep. 2018-02-12.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Sato, S., Uchihara, T., et al. Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice. Sci Rep. 2018-02-12.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Sato, S., Uchihara, T., et al. Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice. Sci Rep. 2018-02-12.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Sato, S., Uchihara, T., et al. Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice. Sci Rep. 2018-02-12.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Sato, S., Uchihara, T., et al. Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice. Sci Rep. 2018-02-12.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Sato, S., Uchihara, T., et al. Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice. Sci Rep. 2018-02-12.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

AMPK Inhibits ULK1-Dependent Autophagosome Formation and Lysosomal Acidification via Distinct Mechanisms.

Nwadike, C., Williamson, L. E., et al. AMPK Inhibits ULK1-Dependent Autophagosome Formation and Lysosomal Acidification via Distinct Mechanisms. Mol Cell Biol. 2018-05-15.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Live-Cell Mesothelioma Biobank to Explore Mechanisms of Tumor Progression.

Oehl, K., Kresoja-Rakic, J., et al. Live-Cell Mesothelioma Biobank to Explore Mechanisms of Tumor Progression. Front Oncol. 2018-03-13.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Mfn2 ubiquitination by PINK1/parkin gates the p97-dependent release of ER from mitochondria to drive mitophagy.

McLelland, G. L., Goiran, T., et al. Mfn2 ubiquitination by PINK1/parkin gates the p97-dependent release of ER from mitochondria to drive mitophagy. Elife. 2018-04-20.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Mfn2 ubiquitination by PINK1/parkin gates the p97-dependent release of ER from mitochondria to drive mitophagy.

McLelland, G. L., Goiran, T., et al. Mfn2 ubiquitination by PINK1/parkin gates the p97-dependent release of ER from mitochondria to drive mitophagy. Elife. 2018-04-20.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Liu, S., Li, Y., et al. Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis. Cell Death Dis. 2018-05-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Liu, S., Li, Y., et al. Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis. Cell Death Dis. 2018-05-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Liu, S., Li, Y., et al. Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis. Cell Death Dis. 2018-05-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Liu, S., Li, Y., et al. Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis. Cell Death Dis. 2018-05-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Liu, S., Li, Y., et al. Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis. Cell Death Dis. 2018-05-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Liu, S., Li, Y., et al. Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis. Cell Death Dis. 2018-05-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Liu, S., Li, Y., et al. Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis. Cell Death Dis. 2018-05-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Liu, S., Li, Y., et al. Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis. Cell Death Dis. 2018-05-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Systemic overexpression of SQSTM1/p62 accelerates disease onset in a SOD1H46R-expressing ALS mouse model.

Mitsui, S., Otomo, A., et al. Systemic overexpression of SQSTM1/p62 accelerates disease onset in a SOD1H46R-expressing ALS mouse model. Mol Brain. 2018-05-29.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-paraffin-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Systemic overexpression of SQSTM1/p62 accelerates disease onset in a SOD1H46R-expressing ALS mouse model.

Mitsui, S., Otomo, A., et al. Systemic overexpression of SQSTM1/p62 accelerates disease onset in a SOD1H46R-expressing ALS mouse model. Mol Brain. 2018-05-29.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-paraffin-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Systemic overexpression of SQSTM1/p62 accelerates disease onset in a SOD1H46R-expressing ALS mouse model.

Mitsui, S., Otomo, A., et al. Systemic overexpression of SQSTM1/p62 accelerates disease onset in a SOD1H46R-expressing ALS mouse model. Mol Brain. 2018-05-29.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-paraffin-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

The ubiquitin ligase UBR5 suppresses proteostasis collapse in pluripotent stem cells from Huntington's disease patients.

Koyuncu, S., Saez, I., et al. The ubiquitin ligase UBR5 suppresses proteostasis collapse in pluripotent stem cells from Huntington's disease patients. Nat Commun. 2018-07-23.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy mediates epithelial cancer chemoresistance by reducing p62/SQSTM1 accumulation.

Battista, R. A., Resnati, M., et al. Autophagy mediates epithelial cancer chemoresistance by reducing p62/SQSTM1 accumulation. PLoS One. 2018-08-02.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy mediates epithelial cancer chemoresistance by reducing p62/SQSTM1 accumulation.

Battista, R. A., Resnati, M., et al. Autophagy mediates epithelial cancer chemoresistance by reducing p62/SQSTM1 accumulation. PLoS One. 2018-08-02.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy mediates epithelial cancer chemoresistance by reducing p62/SQSTM1 accumulation.

Battista, R. A., Resnati, M., et al. Autophagy mediates epithelial cancer chemoresistance by reducing p62/SQSTM1 accumulation. PLoS One. 2018-08-02.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy mediates epithelial cancer chemoresistance by reducing p62/SQSTM1 accumulation.

Battista, R. A., Resnati, M., et al. Autophagy mediates epithelial cancer chemoresistance by reducing p62/SQSTM1 accumulation. PLoS One. 2018-08-02.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

NADPH Oxidase and Guanylate Binding Protein 5 Restrict Survival of Avirulent Type III Strains of Toxoplasma gondii in Naive Macrophages.

Matta, S. K., Patten, K., et al. NADPH Oxidase and Guanylate Binding Protein 5 Restrict Survival of Avirulent Type III Strains of Toxoplasma gondii in Naive Macrophages. MBio. 2018-08-28.

Species/Reactant: Toxoplasma gondii
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

The PPARα Agonist Fenofibrate Prevents Formation of Protein Aggregates (Mallory-Denk bodies) in a Murine Model of Steatohepatitis-like Hepatotoxicity.

Nikam, A., Patankar, J. V., et al. The PPARα Agonist Fenofibrate Prevents Formation of Protein Aggregates (Mallory-Denk bodies) in a Murine Model of Steatohepatitis-like Hepatotoxicity. Sci Rep. 2018-08-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

The PPARα Agonist Fenofibrate Prevents Formation of Protein Aggregates (Mallory-Denk bodies) in a Murine Model of Steatohepatitis-like Hepatotoxicity.

Nikam, A., Patankar, J. V., et al. The PPARα Agonist Fenofibrate Prevents Formation of Protein Aggregates (Mallory-Denk bodies) in a Murine Model of Steatohepatitis-like Hepatotoxicity. Sci Rep. 2018-08-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

The PPARα Agonist Fenofibrate Prevents Formation of Protein Aggregates (Mallory-Denk bodies) in a Murine Model of Steatohepatitis-like Hepatotoxicity.

Nikam, A., Patankar, J. V., et al. The PPARα Agonist Fenofibrate Prevents Formation of Protein Aggregates (Mallory-Denk bodies) in a Murine Model of Steatohepatitis-like Hepatotoxicity. Sci Rep. 2018-08-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

The PPARα Agonist Fenofibrate Prevents Formation of Protein Aggregates (Mallory-Denk bodies) in a Murine Model of Steatohepatitis-like Hepatotoxicity.

Nikam, A., Patankar, J. V., et al. The PPARα Agonist Fenofibrate Prevents Formation of Protein Aggregates (Mallory-Denk bodies) in a Murine Model of Steatohepatitis-like Hepatotoxicity. Sci Rep. 2018-08-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

A short isoform of ATG7 fails to lipidate LC3/GABARAP.

Ogmundsdóttir, M. H., Fock, V., et al. A short isoform of ATG7 fails to lipidate LC3/GABARAP. Sci Rep. 2018-09-26.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Deubiquitinase Usp12 functions noncatalytically to induce autophagy and confer neuroprotection in models of Huntington's disease.

Aron, R., Pellegrini, P., et al. Deubiquitinase Usp12 functions noncatalytically to induce autophagy and confer neuroprotection in models of Huntington's disease. Nat Commun. 2018-09-28.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Resveratrol Ameliorates Mitophagy Disturbance and Improves Cardiac Pathophysiology of Dystrophin-deficient mdx Mice.

Kuno, A., Hosoda, R., et al. Resveratrol Ameliorates Mitophagy Disturbance and Improves Cardiac Pathophysiology of Dystrophin-deficient mdx Mice. Sci Rep. 2018-10-22.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Resveratrol Ameliorates Mitophagy Disturbance and Improves Cardiac Pathophysiology of Dystrophin-deficient mdx Mice.

Kuno, A., Hosoda, R., et al. Resveratrol Ameliorates Mitophagy Disturbance and Improves Cardiac Pathophysiology of Dystrophin-deficient mdx Mice. Sci Rep. 2018-10-22.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Takahashi, M., Kitaura, H., et al. USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis. iScience. 2018-11-30.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Resveratrol Decreases Oxidative Stress by Restoring Mitophagy and Improves the Pathophysiology of Dystrophin-Deficient mdx Mice.

Sebori, R., Kuno, A., et al. Resveratrol Decreases Oxidative Stress by Restoring Mitophagy and Improves the Pathophysiology of Dystrophin-Deficient mdx Mice. Oxid Med Cell Longev. 2018-12-05.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Resveratrol Decreases Oxidative Stress by Restoring Mitophagy and Improves the Pathophysiology of Dystrophin-Deficient mdx Mice.

Sebori, R., Kuno, A., et al. Resveratrol Decreases Oxidative Stress by Restoring Mitophagy and Improves the Pathophysiology of Dystrophin-Deficient mdx Mice. Oxid Med Cell Longev. 2018-12-05.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

RGS9-2 rescues dopamine D2 receptor levels and signaling in DYT1 dystonia mouse models.

Bonsi, P., Ponterio, G., et al. RGS9-2 rescues dopamine D2 receptor levels and signaling in DYT1 dystonia mouse models. EMBO Mol Med. 2019-01-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy attenuates tubulointerstital fibrosis through regulating transforming growth factor-β and NLRP3 inflammasome signaling pathway.

Nam, S. A., Kim, W. Y., et al. Autophagy attenuates tubulointerstital fibrosis through regulating transforming growth factor-β and NLRP3 inflammasome signaling pathway. Cell Death Dis. 2019-01-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells.

Wang, Y., Xiong, H., et al. Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells. Autophagy. 2019-05-01.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells.

Wang, Y., Xiong, H., et al. Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells. Autophagy. 2019-05-01.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells.

Wang, Y., Xiong, H., et al. Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells. Autophagy. 2019-05-01.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells.

Wang, Y., Xiong, H., et al. Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells. Autophagy. 2019-05-01.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Atg7-dependent canonical autophagy regulates the degradation of aquaporin 2 in prolonged hypokalemia.

Kim, W. Y., Nam, S. A., et al. Atg7-dependent canonical autophagy regulates the degradation of aquaporin 2 in prolonged hypokalemia. Sci Rep. 2019-02-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Atg7-dependent canonical autophagy regulates the degradation of aquaporin 2 in prolonged hypokalemia.

Kim, W. Y., Nam, S. A., et al. Atg7-dependent canonical autophagy regulates the degradation of aquaporin 2 in prolonged hypokalemia. Sci Rep. 2019-02-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-paraffin

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Atg7-dependent canonical autophagy regulates the degradation of aquaporin 2 in prolonged hypokalemia.

Kim, W. Y., Nam, S. A., et al. Atg7-dependent canonical autophagy regulates the degradation of aquaporin 2 in prolonged hypokalemia. Sci Rep. 2019-02-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Brain tyrosinase overexpression implicates age-dependent neuromelanin production in Parkinson's disease pathogenesis.

Carballo-Carbajal, I., Laguna, A., et al. Brain tyrosinase overexpression implicates age-dependent neuromelanin production in Parkinson's disease pathogenesis. Nat Commun. 2019-03-07.

Species/Reactant: Rattus norvegicus (Rat)
Applications: Immunohistochemistry-paraffin-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Brain tyrosinase overexpression implicates age-dependent neuromelanin production in Parkinson's disease pathogenesis.

Carballo-Carbajal, I., Laguna, A., et al. Brain tyrosinase overexpression implicates age-dependent neuromelanin production in Parkinson's disease pathogenesis. Nat Commun. 2019-03-07.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Myoglobinopathy is an adult-onset autosomal dominant myopathy with characteristic sarcoplasmic inclusions.

Olive, M., Engvall, M., et al. Myoglobinopathy is an adult-onset autosomal dominant myopathy with characteristic sarcoplasmic inclusions. Nat Commun. 2019-03-27.

Species/Reactant: Homo sapiens (Human)
Applications: Immunohistochemistry-frozen tissue

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Saito, T., Kuma, A., et al. Autophagy regulates lipid metabolism through selective turnover of NCoR1. Nat Commun. 2019-04-05.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Saito, T., Kuma, A., et al. Autophagy regulates lipid metabolism through selective turnover of NCoR1. Nat Commun. 2019-04-05.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Saito, T., Kuma, A., et al. Autophagy regulates lipid metabolism through selective turnover of NCoR1. Nat Commun. 2019-04-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Saito, T., Kuma, A., et al. Autophagy regulates lipid metabolism through selective turnover of NCoR1. Nat Commun. 2019-04-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Saito, T., Kuma, A., et al. Autophagy regulates lipid metabolism through selective turnover of NCoR1. Nat Commun. 2019-04-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Saito, T., Kuma, A., et al. Autophagy regulates lipid metabolism through selective turnover of NCoR1. Nat Commun. 2019-04-05.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Saito, T., Kuma, A., et al. Autophagy regulates lipid metabolism through selective turnover of NCoR1. Nat Commun. 2019-04-05.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Saito, T., Kuma, A., et al. Autophagy regulates lipid metabolism through selective turnover of NCoR1. Nat Commun. 2019-04-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Saito, T., Kuma, A., et al. Autophagy regulates lipid metabolism through selective turnover of NCoR1. Nat Commun. 2019-04-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Saito, T., Kuma, A., et al. Autophagy regulates lipid metabolism through selective turnover of NCoR1. Nat Commun. 2019-04-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

The FMRpolyGlycine Protein Mediates Aggregate Formation and Toxicity Independent of the CGG mRNA Hairpin in a Cellular Model for FXTAS.

Hoem, G., Bowitz Larsen, K., et al. The FMRpolyGlycine Protein Mediates Aggregate Formation and Toxicity Independent of the CGG mRNA Hairpin in a Cellular Model for FXTAS. Front Genet. 2019-04-16.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Hyperosmotic Stress Induces Unconventional Autophagy Independent of the Ulk1 Complex.

Tamura, N., Kageyama, S., et al. Hyperosmotic Stress Induces Unconventional Autophagy Independent of the Ulk1 Complex. Mol Cell Biol. 2019-08-15.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Hyperosmotic Stress Induces Unconventional Autophagy Independent of the Ulk1 Complex.

Tamura, N., Kageyama, S., et al. Hyperosmotic Stress Induces Unconventional Autophagy Independent of the Ulk1 Complex. Mol Cell Biol. 2019-08-15.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Hyperosmotic Stress Induces Unconventional Autophagy Independent of the Ulk1 Complex.

Tamura, N., Kageyama, S., et al. Hyperosmotic Stress Induces Unconventional Autophagy Independent of the Ulk1 Complex. Mol Cell Biol. 2019-08-15.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Hyperosmotic Stress Induces Unconventional Autophagy Independent of the Ulk1 Complex.

Tamura, N., Kageyama, S., et al. Hyperosmotic Stress Induces Unconventional Autophagy Independent of the Ulk1 Complex. Mol Cell Biol. 2019-08-15.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

m6A mRNA demethylase FTO regulates melanoma tumorigenicity and response to anti-PD-1 blockade.

Yang, S., Wei, J., et al. m6A mRNA demethylase FTO regulates melanoma tumorigenicity and response to anti-PD-1 blockade. Nat Commun. 2019-06-25.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells.

Riemschoss, K., Arndt, V., et al. Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells. Life Sci Alliance. 2019-08-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells.

Riemschoss, K., Arndt, V., et al. Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells. Life Sci Alliance. 2019-08-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells.

Riemschoss, K., Arndt, V., et al. Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells. Life Sci Alliance. 2019-08-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells.

Riemschoss, K., Arndt, V., et al. Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells. Life Sci Alliance. 2019-08-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells.

Riemschoss, K., Arndt, V., et al. Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells. Life Sci Alliance. 2019-08-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells.

Riemschoss, K., Arndt, V., et al. Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells. Life Sci Alliance. 2019-08-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins.

Lim, J., Park, H., et al. Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins. Elife. 2019-07-09.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins.

Lim, J., Park, H., et al. Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins. Elife. 2019-07-09.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins.

Lim, J., Park, H., et al. Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins. Elife. 2019-07-09.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins.

Lim, J., Park, H., et al. Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins. Elife. 2019-07-09.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins.

Lim, J., Park, H., et al. Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins. Elife. 2019-07-09.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

cPLA2 activation contributes to lysosomal defects leading to impairment of autophagy after spinal cord injury.

Li, Y., Jones, J. W., et al. cPLA2 activation contributes to lysosomal defects leading to impairment of autophagy after spinal cord injury. Cell Death Dis. 2019-07-11.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4.

Castets, P., Rion, N., et al. mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4. Nat Commun. 2019-07-18.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4.

Castets, P., Rion, N., et al. mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4. Nat Commun. 2019-07-18.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4.

Castets, P., Rion, N., et al. mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4. Nat Commun. 2019-07-18.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-paraffin

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4.

Castets, P., Rion, N., et al. mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4. Nat Commun. 2019-07-18.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4.

Castets, P., Rion, N., et al. mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4. Nat Commun. 2019-07-18.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Lysosomal degradation of newly formed insulin granules contributes to β cell failure in diabetes.

Pasquier, A., Vivot, K., et al. Lysosomal degradation of newly formed insulin granules contributes to β cell failure in diabetes. Nat Commun. 2019-07-25.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning.

Pellegrini, C., Columbaro, M., et al. Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning. Exp Mol Med. 2019-08-02.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning.

Pellegrini, C., Columbaro, M., et al. Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning. Exp Mol Med. 2019-08-02.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning.

Pellegrini, C., Columbaro, M., et al. Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning. Exp Mol Med. 2019-08-02.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning.

Pellegrini, C., Columbaro, M., et al. Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning. Exp Mol Med. 2019-08-02.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Hill, C., Li, J., et al. Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis. Cell Death Dis. 2019-08-07.

Species/Reactant: Homo sapiens (Human)
Applications: Immunohistochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Hill, C., Li, J., et al. Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis. Cell Death Dis. 2019-08-07.

Species/Reactant: Homo sapiens (Human)
Applications: Immunohistochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Hill, C., Li, J., et al. Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis. Cell Death Dis. 2019-08-07.

Species/Reactant: Homo sapiens (Human)
Applications: Immunohistochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Hill, C., Li, J., et al. Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis. Cell Death Dis. 2019-08-07.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Hill, C., Li, J., et al. Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis. Cell Death Dis. 2019-08-07.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Hill, C., Li, J., et al. Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis. Cell Death Dis. 2019-08-07.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Hill, C., Li, J., et al. Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis. Cell Death Dis. 2019-08-07.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Hill, C., Li, J., et al. Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis. Cell Death Dis. 2019-08-07.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Yang, Y., Willis, T. L., et al. Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response. Nat Commun. 2019-08-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Yang, Y., Willis, T. L., et al. Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response. Nat Commun. 2019-08-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Yang, Y., Willis, T. L., et al. Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response. Nat Commun. 2019-08-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Yang, Y., Willis, T. L., et al. Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response. Nat Commun. 2019-08-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Yang, Y., Willis, T. L., et al. Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response. Nat Commun. 2019-08-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Yang, Y., Willis, T. L., et al. Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response. Nat Commun. 2019-08-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Yang, Y., Willis, T. L., et al. Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response. Nat Commun. 2019-08-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Buffering of cytosolic calcium plays a neuroprotective role by preserving the autophagy-lysosome pathway during MPP+-induced neuronal death.

Jung, S., Chung, Y., et al. Buffering of cytosolic calcium plays a neuroprotective role by preserving the autophagy-lysosome pathway during MPP+-induced neuronal death. Cell Death Discov. 2019-08-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Buffering of cytosolic calcium plays a neuroprotective role by preserving the autophagy-lysosome pathway during MPP+-induced neuronal death.

Jung, S., Chung, Y., et al. Buffering of cytosolic calcium plays a neuroprotective role by preserving the autophagy-lysosome pathway during MPP+-induced neuronal death. Cell Death Discov. 2019-08-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Buffering of cytosolic calcium plays a neuroprotective role by preserving the autophagy-lysosome pathway during MPP+-induced neuronal death.

Jung, S., Chung, Y., et al. Buffering of cytosolic calcium plays a neuroprotective role by preserving the autophagy-lysosome pathway during MPP+-induced neuronal death. Cell Death Discov. 2019-08-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Buffering of cytosolic calcium plays a neuroprotective role by preserving the autophagy-lysosome pathway during MPP+-induced neuronal death.

Jung, S., Chung, Y., et al. Buffering of cytosolic calcium plays a neuroprotective role by preserving the autophagy-lysosome pathway during MPP+-induced neuronal death. Cell Death Discov. 2019-08-28.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

Anisimov, S., Takahashi, M., et al. G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10. Sci Rep. 2019-09-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

Anisimov, S., Takahashi, M., et al. G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10. Sci Rep. 2019-09-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

Anisimov, S., Takahashi, M., et al. G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10. Sci Rep. 2019-09-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

Anisimov, S., Takahashi, M., et al. G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10. Sci Rep. 2019-09-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

Anisimov, S., Takahashi, M., et al. G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10. Sci Rep. 2019-09-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

Anisimov, S., Takahashi, M., et al. G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10. Sci Rep. 2019-09-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

Anisimov, S., Takahashi, M., et al. G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10. Sci Rep. 2019-09-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

Anisimov, S., Takahashi, M., et al. G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10. Sci Rep. 2019-09-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

Anisimov, S., Takahashi, M., et al. G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10. Sci Rep. 2019-09-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

Anisimov, S., Takahashi, M., et al. G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10. Sci Rep. 2019-09-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

Anisimov, S., Takahashi, M., et al. G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10. Sci Rep. 2019-09-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

Anisimov, S., Takahashi, M., et al. G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10. Sci Rep. 2019-09-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Schwob, A., Teruel, É., et al. SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function. Sci Rep. 2019-11-05.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy protein NRBF2 has reduced expression in Alzheimer's brains and modulates memory and amyloid-beta homeostasis in mice.

Lachance, V., Wang, Q., et al. Autophagy protein NRBF2 has reduced expression in Alzheimer's brains and modulates memory and amyloid-beta homeostasis in mice. Mol Neurodegener. 2019-11-27.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy protein NRBF2 has reduced expression in Alzheimer's brains and modulates memory and amyloid-beta homeostasis in mice.

Lachance, V., Wang, Q., et al. Autophagy protein NRBF2 has reduced expression in Alzheimer's brains and modulates memory and amyloid-beta homeostasis in mice. Mol Neurodegener. 2019-11-27.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of autophagy impairs physiological steatosis by accumulation of NCoR1.

Takahashi, S. S., Sou, Y. S., et al. Loss of autophagy impairs physiological steatosis by accumulation of NCoR1. Life Sci Alliance. 2020-01-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of autophagy impairs physiological steatosis by accumulation of NCoR1.

Takahashi, S. S., Sou, Y. S., et al. Loss of autophagy impairs physiological steatosis by accumulation of NCoR1. Life Sci Alliance. 2020-01-01.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of autophagy impairs physiological steatosis by accumulation of NCoR1.

Takahashi, S. S., Sou, Y. S., et al. Loss of autophagy impairs physiological steatosis by accumulation of NCoR1. Life Sci Alliance. 2020-01-01.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy Deficiency in Renal Proximal Tubular Cells Leads to an Increase in Cellular Injury and Apoptosis under Normal Fed Conditions.

Suzuki, C., Tanida, I., et al. Autophagy Deficiency in Renal Proximal Tubular Cells Leads to an Increase in Cellular Injury and Apoptosis under Normal Fed Conditions. Int J Mol Sci. 2019-12-25.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy Deficiency in Renal Proximal Tubular Cells Leads to an Increase in Cellular Injury and Apoptosis under Normal Fed Conditions.

Suzuki, C., Tanida, I., et al. Autophagy Deficiency in Renal Proximal Tubular Cells Leads to an Increase in Cellular Injury and Apoptosis under Normal Fed Conditions. Int J Mol Sci. 2019-12-25.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy Deficiency in Renal Proximal Tubular Cells Leads to an Increase in Cellular Injury and Apoptosis under Normal Fed Conditions.

Suzuki, C., Tanida, I., et al. Autophagy Deficiency in Renal Proximal Tubular Cells Leads to an Increase in Cellular Injury and Apoptosis under Normal Fed Conditions. Int J Mol Sci. 2019-12-25.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

AMPK-dependent activation of the Cyclin Y/CDK16 complex controls autophagy.

Dohmen, M., Krieg, S., et al. AMPK-dependent activation of the Cyclin Y/CDK16 complex controls autophagy. Nat Commun. 2020-02-25.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

AMPK-dependent activation of the Cyclin Y/CDK16 complex controls autophagy.

Dohmen, M., Krieg, S., et al. AMPK-dependent activation of the Cyclin Y/CDK16 complex controls autophagy. Nat Commun. 2020-02-25.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

AMPK-dependent activation of the Cyclin Y/CDK16 complex controls autophagy.

Dohmen, M., Krieg, S., et al. AMPK-dependent activation of the Cyclin Y/CDK16 complex controls autophagy. Nat Commun. 2020-02-25.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Choi, I., Zhang, Y., et al. Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration. Nat Commun. 2020-03-13.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Myocardial cathepsin D is downregulated in sudden cardiac death.

Kakimoto, Y., Sasaki, A., et al. Myocardial cathepsin D is downregulated in sudden cardiac death. PLoS One. 2020-03-17.

Species/Reactant: Homo sapiens (Human)
Applications: Simple Western - Size

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination.

Blázquez, C., Ruiz-Calvo, A., et al. Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination. Elife. 2020-08-10.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination.

Blázquez, C., Ruiz-Calvo, A., et al. Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination. Elife. 2020-08-10.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination.

Blázquez, C., Ruiz-Calvo, A., et al. Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination. Elife. 2020-08-10.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination.

Blázquez, C., Ruiz-Calvo, A., et al. Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination. Elife. 2020-08-10.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination.

Blázquez, C., Ruiz-Calvo, A., et al. Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination. Elife. 2020-08-10.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination.

Blázquez, C., Ruiz-Calvo, A., et al. Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination. Elife. 2020-08-10.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

A Scaffold-Free 3-D Co-Culture Mimics the Major Features of the Reverse Warburg Effect In Vitro.

Keller, F., Bruch, R., et al. A Scaffold-Free 3-D Co-Culture Mimics the Major Features of the Reverse Warburg Effect In Vitro. Cells. 2020-08-13.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

A Scaffold-Free 3-D Co-Culture Mimics the Major Features of the Reverse Warburg Effect In Vitro.

Keller, F., Bruch, R., et al. A Scaffold-Free 3-D Co-Culture Mimics the Major Features of the Reverse Warburg Effect In Vitro. Cells. 2020-08-13.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

ISG15 Connects Autophagy and IFN-γ-Dependent Control of Toxoplasma gondii Infection in Human Cells.

Bhushan, J., Radke, J. B., et al. ISG15 Connects Autophagy and IFN-γ-Dependent Control of Toxoplasma gondii Infection in Human Cells. MBio. 2020-10-06.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

ISG15 Connects Autophagy and IFN-γ-Dependent Control of Toxoplasma gondii Infection in Human Cells.

Bhushan, J., Radke, J. B., et al. ISG15 Connects Autophagy and IFN-γ-Dependent Control of Toxoplasma gondii Infection in Human Cells. MBio. 2020-10-06.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Baeken, M. W., Weckmann, K., et al. Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C. Cells. 2020-10-18.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Baeken, M. W., Weckmann, K., et al. Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C. Cells. 2020-10-18.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Baeken, M. W., Weckmann, K., et al. Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C. Cells. 2020-10-18.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Baeken, M. W., Weckmann, K., et al. Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C. Cells. 2020-10-18.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Baeken, M. W., Weckmann, K., et al. Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C. Cells. 2020-10-18.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Baeken, M. W., Weckmann, K., et al. Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C. Cells. 2020-10-18.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Baeken, M. W., Weckmann, K., et al. Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C. Cells. 2020-10-18.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Baeken, M. W., Weckmann, K., et al. Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C. Cells. 2020-10-18.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

The Polyphenol Pterostilbene Ameliorates the Myopathic Phenotype of Collagen VI Deficient Mice via Autophagy Induction.

Metti, S., Gambarotto, L., et al. The Polyphenol Pterostilbene Ameliorates the Myopathic Phenotype of Collagen VI Deficient Mice via Autophagy Induction. Front Cell Dev Biol. 2020-11-03.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Degradation of connexin 50 protein causes waterclefts in human lens.

Nakazawa, Y., Shibata, T., et al. Degradation of connexin 50 protein causes waterclefts in human lens. Open Med (Wars). 2020-12-19.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment.

Berning, L., Schlütermann, D., et al. Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment. Molecules. 2021-02-27.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

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Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment.

Berning, L., Schlütermann, D., et al. Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment. Molecules. 2021-02-27.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment.

Berning, L., Schlütermann, D., et al. Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment. Molecules. 2021-02-27.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment.

Berning, L., Schlütermann, D., et al. Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment. Molecules. 2021-02-27.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment.

Berning, L., Schlütermann, D., et al. Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment. Molecules. 2021-02-27.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis.

Cui, Y. H., Yang, S., et al. Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis. Nat Commun. 2021-04-12.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis.

Cui, Y. H., Yang, S., et al. Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis. Nat Commun. 2021-04-12.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis.

Cui, Y. H., Yang, S., et al. Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis. Nat Commun. 2021-04-12.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis.

Cui, Y. H., Yang, S., et al. Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis. Nat Commun. 2021-04-12.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

N-Acetyl-L-leucine improves functional recovery and attenuates cortical cell death and neuroinflammation after traumatic brain injury in mice.

Hegdekar, N., Lipinski, M. M., et al. N-Acetyl-L-leucine improves functional recovery and attenuates cortical cell death and neuroinflammation after traumatic brain injury in mice. Sci Rep. 2021-04-29.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

High-throughput screening for natural compound-based autophagy modulators reveals novel chemotherapeutic mode of action for arzanol.

Deitersen, J., Berning, L., et al. High-throughput screening for natural compound-based autophagy modulators reveals novel chemotherapeutic mode of action for arzanol. Cell Death Dis. 2021-05-31.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

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High-throughput screening for natural compound-based autophagy modulators reveals novel chemotherapeutic mode of action for arzanol.

Deitersen, J., Berning, L., et al. High-throughput screening for natural compound-based autophagy modulators reveals novel chemotherapeutic mode of action for arzanol. Cell Death Dis. 2021-05-31.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

An essential role of the autophagy activating kinase ULK1 in snRNP biogenesis.

Schmitz, K., Cox, J., et al. An essential role of the autophagy activating kinase ULK1 in snRNP biogenesis. Nucleic Acids Res. 2021-06-21.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Mercer, T. J., Ohashi, Y., et al. Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy. EMBO J. 2021-07-15.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Mercer, T. J., Ohashi, Y., et al. Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy. EMBO J. 2021-07-15.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Mercer, T. J., Ohashi, Y., et al. Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy. EMBO J. 2021-07-15.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Mercer, T. J., Ohashi, Y., et al. Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy. EMBO J. 2021-07-15.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Mercer, T. J., Ohashi, Y., et al. Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy. EMBO J. 2021-07-15.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Mercer, T. J., Ohashi, Y., et al. Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy. EMBO J. 2021-07-15.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Mercer, T. J., Ohashi, Y., et al. Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy. EMBO J. 2021-07-15.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Mercer, T. J., Ohashi, Y., et al. Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy. EMBO J. 2021-07-15.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Different Roles of p62 (SQSTM1) Isoforms in Keratin-Related Protein Aggregation.

Somlapura, M., Gottschalk, B., et al. Different Roles of p62 (SQSTM1) Isoforms in Keratin-Related Protein Aggregation. Int J Mol Sci. 2021-06-09.

Species/Reactant: Homo sapiens (Human)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Different Roles of p62 (SQSTM1) Isoforms in Keratin-Related Protein Aggregation.

Somlapura, M., Gottschalk, B., et al. Different Roles of p62 (SQSTM1) Isoforms in Keratin-Related Protein Aggregation. Int J Mol Sci. 2021-06-09.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Different Roles of p62 (SQSTM1) Isoforms in Keratin-Related Protein Aggregation.

Somlapura, M., Gottschalk, B., et al. Different Roles of p62 (SQSTM1) Isoforms in Keratin-Related Protein Aggregation. Int J Mol Sci. 2021-06-09.

Species/Reactant: Homo sapiens (Human)
Applications: Immunocytochemistry-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

FIP200 controls the TBK1 activation threshold at SQSTM1/p62-positive condensates.

Schlütermann, D., Berleth, N., et al. FIP200 controls the TBK1 activation threshold at SQSTM1/p62-positive condensates. Sci Rep. 2021-07-05.

Species/Reactant: Mus musculus (House mouse)
Applications: Western Blotting

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Lack of Cathepsin D in the central nervous system results in microglia and astrocyte activation and the accumulation of proteinopathy-related proteins.

Suzuki, C., Yamaguchi, J., et al. Lack of Cathepsin D in the central nervous system results in microglia and astrocyte activation and the accumulation of proteinopathy-related proteins. Sci Rep. 2022-07-08.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Malin restoration as proof of concept for gene therapy for Lafora disease.

Varea, O., Guinovart, J. J., et al. Malin restoration as proof of concept for gene therapy for Lafora disease. Brain Commun. 2022-07-12.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry-paraffin-immunofluorescence

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

BALB.NCT-Cpox nct is a unique mouse model of hereditary coproporphyria.

Kang, X., Shimada, S., et al. BALB.NCT-Cpox nct is a unique mouse model of hereditary coproporphyria. Mol Genet Metab Rep. 2023-06-01.

Species/Reactant: Mus musculus (House mouse)
Applications: Immunohistochemistry

Image collected and cropped by CiteAb from the following publication, provided under a CC-BY licence.

Loss of ALS2/Alsin exacerbates motor dysfunction in a SOD1-expressing mouse ALS model by disturbing endolysosomal trafficking.

Loss of ALS2/Alsin exacerbates motor dysfunction in a SOD1-expressing mouse ALS model by disturbing endolysosomal trafficking.

Inhibition of mTOR by rapamycin abolishes cognitive deficits and reduces amyloid-beta levels in a mouse model of Alzheimer's disease.

Targeting protective autophagy exacerbates UV-triggered apoptotic cell death.

Cell loss and autophagy in the extra-adrenal chromaffin organ of Zuckerkandl are regulated by glucocorticoid signalling.

Cell loss and autophagy in the extra-adrenal chromaffin organ of Zuckerkandl are regulated by glucocorticoid signalling.

Rapamycin attenuates the progression of tau pathology in P301S tau transgenic mice.

A novel acylaminoimidazole derivative, WN1316, alleviates disease progression via suppression of glial inflammation in ALS mouse model.

A novel acylaminoimidazole derivative, WN1316, alleviates disease progression via suppression of glial inflammation in ALS mouse model.

Rotavirus increases levels of lipidated LC3 supporting accumulation of infectious progeny virus without inducing autophagosome formation.

Rotavirus increases levels of lipidated LC3 supporting accumulation of infectious progeny virus without inducing autophagosome formation.

Ribosomal protein mutations induce autophagy through S6 kinase inhibition of the insulin pathway.

Ribosomal protein mutations induce autophagy through S6 kinase inhibition of the insulin pathway.

Ribosomal protein mutations induce autophagy through S6 kinase inhibition of the insulin pathway.

Ribosomal protein mutations induce autophagy through S6 kinase inhibition of the insulin pathway.

Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death.

Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death.

Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death.

Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death.

Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death.

Disrupted autophagy after spinal cord injury is associated with ER stress and neuronal cell death.

Cucurbitacin E has neuroprotective properties and autophagic modulating activities on dopaminergic neurons.

The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates.

The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates.

The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates.

The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates.

The unexpected role of polyubiquitin chains in the formation of fibrillar aggregates.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Proteotoxic stress induces phosphorylation of p62/SQSTM1 by ULK1 to regulate selective autophagic clearance of protein aggregates.

Autophagic signaling and proteolytic enzyme activity in cardiac and skeletal muscle of spontaneously hypertensive rats following chronic aerobic exercise.

Estrogen receptor α regulates non-canonical autophagy that provides stress resistance to neuroblastoma and breast cancer cells and involves BAG3 function.

Frontotemporal dementia caused by CHMP2B mutation is characterised by neuronal lysosomal storage pathology.

Frontotemporal dementia caused by CHMP2B mutation is characterised by neuronal lysosomal storage pathology.

Frontotemporal dementia caused by CHMP2B mutation is characterised by neuronal lysosomal storage pathology.

Frontotemporal dementia caused by CHMP2B mutation is characterised by neuronal lysosomal storage pathology.

All-trans retinoic acid and rapamycin normalize Hutchinson Gilford progeria fibroblast phenotype.

Sqstm1-GFP knock-in mice reveal dynamic actions of Sqstm1 during autophagy and under stress conditions in living cells.

Sqstm1-GFP knock-in mice reveal dynamic actions of Sqstm1 during autophagy and under stress conditions in living cells.

Sqstm1-GFP knock-in mice reveal dynamic actions of Sqstm1 during autophagy and under stress conditions in living cells.

Sqstm1-GFP knock-in mice reveal dynamic actions of Sqstm1 during autophagy and under stress conditions in living cells.

Autophagy and endosomal trafficking inhibition by Vibrio cholerae MARTX toxin phosphatidylinositol-3-phosphate-specific phospholipase A1 activity.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

Identification of p62/SQSTM1 as a component of non-canonical Wnt VANGL2-JNK signalling in breast cancer.

mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy.

mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy.

mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy.

mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy.

mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy.

mTOR Hyperactivation by Ablation of Tuberous Sclerosis Complex 2 in the Mouse Heart Induces Cardiac Dysfunction with the Increased Number of Small Mitochondria Mediated through the Down-Regulation of Autophagy.

A Founder Mutation in VPS11 Causes an Autosomal Recessive Leukoencephalopathy Linked to Autophagic Defects.

p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming.

p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming.

p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming.

p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming.

p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming.

p62/Sqstm1 promotes malignancy of HCV-positive hepatocellular carcinoma through Nrf2-dependent metabolic reprogramming.

TSC but not PTEN loss in starving cones of retinitis pigmentosa mice leads to an autophagy defect and mTORC1 dissociation from the lysosome.

TSC but not PTEN loss in starving cones of retinitis pigmentosa mice leads to an autophagy defect and mTORC1 dissociation from the lysosome.

TSC but not PTEN loss in starving cones of retinitis pigmentosa mice leads to an autophagy defect and mTORC1 dissociation from the lysosome.

TSC but not PTEN loss in starving cones of retinitis pigmentosa mice leads to an autophagy defect and mTORC1 dissociation from the lysosome.

Intracellular glutathione determines bortezomib cytotoxicity in multiple myeloma cells.

Effect of Exendin-4 on Autophagy Clearance in Beta Cell of Rats with Tacrolimus-induced Diabetes Mellitus.

Oleuropein Prevents Neuronal Death, Mitigates Mitochondrial Superoxide Production and Modulates Autophagy in a Dopaminergic Cellular Model.

Hydroxychloroquine potentiates carfilzomib toxicity towards myeloma cells.

Hydroxychloroquine potentiates carfilzomib toxicity towards myeloma cells.

Differential roles of NF-Y transcription factor in ER chaperone expression and neuronal maintenance in the CNS.

Differential roles of NF-Y transcription factor in ER chaperone expression and neuronal maintenance in the CNS.

Differential roles of NF-Y transcription factor in ER chaperone expression and neuronal maintenance in the CNS.

Activation of the Keap1/Nrf2 stress response pathway in autophagic vacuolar myopathies.

Activation of the Keap1/Nrf2 stress response pathway in autophagic vacuolar myopathies.

Activation of the Keap1/Nrf2 stress response pathway in autophagic vacuolar myopathies.

Activation of the Keap1/Nrf2 stress response pathway in autophagic vacuolar myopathies.

Autophagy dysregulation in Danon disease.

Autophagy dysregulation in Danon disease.

Autophagy dysregulation in Danon disease.

Gastrin activates autophagy and increases migration and survival of gastric adenocarcinoma cells.

Gastrin activates autophagy and increases migration and survival of gastric adenocarcinoma cells.

Gastrin activates autophagy and increases migration and survival of gastric adenocarcinoma cells.

Gastrin activates autophagy and increases migration and survival of gastric adenocarcinoma cells.

Propagation of pathological α-synuclein in marmoset brain.

Sarcolipin deletion exacerbates soleus muscle atrophy and weakness in phospholamban overexpressing mice.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Exploiting macrophage autophagy-lysosomal biogenesis as a therapy for atherosclerosis.

Impaired autophagosome clearance contributes to neuronal death in a piglet model of neonatal hypoxic-ischemic encephalopathy.

Cryptic amyloidogenic elements in mutant NEFH causing Charcot-Marie-Tooth 2 trigger aggresome formation and neuronal death.

Cryptic amyloidogenic elements in mutant NEFH causing Charcot-Marie-Tooth 2 trigger aggresome formation and neuronal death.

Cryptic amyloidogenic elements in mutant NEFH causing Charcot-Marie-Tooth 2 trigger aggresome formation and neuronal death.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Centriolar Satellites Control GABARAP Ubiquitination and GABARAP-Mediated Autophagy.

Hypoxia ameliorates intestinal inflammation through NLRP3/mTOR downregulation and autophagy activation.

Lysosomotropism depends on glucose: a chloroquine resistance mechanism.

Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells.

Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells.

Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells.

Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells.

Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells.

Selective degradation of PU.1 during autophagy represses the differentiation and antitumour activity of TH9 cells.

Standard Assays for the Study of Autophagy in the Ex Vivo Retina.

Phosphorylation and degradation of αB-crystallin during enterovirus infection facilitates viral replication and induces viral pathogenesis.

L-leucine and SPNS1 coordinately ameliorate dysfunction of autophagy in mouse and human Niemann-Pick type C disease.

L-leucine and SPNS1 coordinately ameliorate dysfunction of autophagy in mouse and human Niemann-Pick type C disease.

Sequestosome 1 Deficiency Delays, but Does Not Prevent Brain Damage Formation Following Acute Brain Injury in Adult Mice.

Sequestosome 1 Deficiency Delays, but Does Not Prevent Brain Damage Formation Following Acute Brain Injury in Adult Mice.

Sequestosome 1 Deficiency Delays, but Does Not Prevent Brain Damage Formation Following Acute Brain Injury in Adult Mice.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Negative Regulation of the Keap1-Nrf2 Pathway by a p62/Sqstm1 Splicing Variant.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Oxidation of SQSTM1/p62 mediates the link between redox state and protein homeostasis.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice.

AMPK Inhibits ULK1-Dependent Autophagosome Formation and Lysosomal Acidification via Distinct Mechanisms.

Live-Cell Mesothelioma Biobank to Explore Mechanisms of Tumor Progression.

Mfn2 ubiquitination by PINK1/parkin gates the p97-dependent release of ER from mitochondria to drive mitophagy.

Mfn2 ubiquitination by PINK1/parkin gates the p97-dependent release of ER from mitochondria to drive mitophagy.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Lysosomal damage after spinal cord injury causes accumulation of RIPK1 and RIPK3 proteins and potentiation of necroptosis.

Systemic overexpression of SQSTM1/p62 accelerates disease onset in a SOD1H46R-expressing ALS mouse model.

Systemic overexpression of SQSTM1/p62 accelerates disease onset in a SOD1H46R-expressing ALS mouse model.

Systemic overexpression of SQSTM1/p62 accelerates disease onset in a SOD1H46R-expressing ALS mouse model.

The ubiquitin ligase UBR5 suppresses proteostasis collapse in pluripotent stem cells from Huntington's disease patients.

Autophagy mediates epithelial cancer chemoresistance by reducing p62/SQSTM1 accumulation.

Autophagy mediates epithelial cancer chemoresistance by reducing p62/SQSTM1 accumulation.

Autophagy mediates epithelial cancer chemoresistance by reducing p62/SQSTM1 accumulation.

Autophagy mediates epithelial cancer chemoresistance by reducing p62/SQSTM1 accumulation.

NADPH Oxidase and Guanylate Binding Protein 5 Restrict Survival of Avirulent Type III Strains of Toxoplasma gondii in Naive Macrophages.

The PPARα Agonist Fenofibrate Prevents Formation of Protein Aggregates (Mallory-Denk bodies) in a Murine Model of Steatohepatitis-like Hepatotoxicity.

The PPARα Agonist Fenofibrate Prevents Formation of Protein Aggregates (Mallory-Denk bodies) in a Murine Model of Steatohepatitis-like Hepatotoxicity.

The PPARα Agonist Fenofibrate Prevents Formation of Protein Aggregates (Mallory-Denk bodies) in a Murine Model of Steatohepatitis-like Hepatotoxicity.

The PPARα Agonist Fenofibrate Prevents Formation of Protein Aggregates (Mallory-Denk bodies) in a Murine Model of Steatohepatitis-like Hepatotoxicity.

A short isoform of ATG7 fails to lipidate LC3/GABARAP.

Deubiquitinase Usp12 functions noncatalytically to induce autophagy and confer neuroprotection in models of Huntington's disease.

Resveratrol Ameliorates Mitophagy Disturbance and Improves Cardiac Pathophysiology of Dystrophin-deficient mdx Mice.

Resveratrol Ameliorates Mitophagy Disturbance and Improves Cardiac Pathophysiology of Dystrophin-deficient mdx Mice.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

USP10 Is a Driver of Ubiquitinated Protein Aggregation and Aggresome Formation to Inhibit Apoptosis.

Resveratrol Decreases Oxidative Stress by Restoring Mitophagy and Improves the Pathophysiology of Dystrophin-Deficient mdx Mice.

Resveratrol Decreases Oxidative Stress by Restoring Mitophagy and Improves the Pathophysiology of Dystrophin-Deficient mdx Mice.

RGS9-2 rescues dopamine D2 receptor levels and signaling in DYT1 dystonia mouse models.

Autophagy attenuates tubulointerstital fibrosis through regulating transforming growth factor-β and NLRP3 inflammasome signaling pathway.

Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells.

Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells.

Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells.

Autophagy inhibition specifically promotes epithelial-mesenchymal transition and invasion in RAS-mutated cancer cells.

Atg7-dependent canonical autophagy regulates the degradation of aquaporin 2 in prolonged hypokalemia.

Atg7-dependent canonical autophagy regulates the degradation of aquaporin 2 in prolonged hypokalemia.

Atg7-dependent canonical autophagy regulates the degradation of aquaporin 2 in prolonged hypokalemia.

Brain tyrosinase overexpression implicates age-dependent neuromelanin production in Parkinson's disease pathogenesis.

Brain tyrosinase overexpression implicates age-dependent neuromelanin production in Parkinson's disease pathogenesis.

Myoglobinopathy is an adult-onset autosomal dominant myopathy with characteristic sarcoplasmic inclusions.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

Autophagy regulates lipid metabolism through selective turnover of NCoR1.

The FMRpolyGlycine Protein Mediates Aggregate Formation and Toxicity Independent of the CGG mRNA Hairpin in a Cellular Model for FXTAS.

Hyperosmotic Stress Induces Unconventional Autophagy Independent of the Ulk1 Complex.

Hyperosmotic Stress Induces Unconventional Autophagy Independent of the Ulk1 Complex.

Hyperosmotic Stress Induces Unconventional Autophagy Independent of the Ulk1 Complex.

Hyperosmotic Stress Induces Unconventional Autophagy Independent of the Ulk1 Complex.

m6A mRNA demethylase FTO regulates melanoma tumorigenicity and response to anti-PD-1 blockade.

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells.

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells.

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells.

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells.

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells.

Fibril-induced glutamine-/asparagine-rich prions recruit stress granule proteins in mammalian cells.

Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins.

Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins.

Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins.

Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins.

Autophagy regulates inflammatory programmed cell death via turnover of RHIM-domain proteins.

cPLA2 activation contributes to lysosomal defects leading to impairment of autophagy after spinal cord injury.

mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4.

mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4.

mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4.

mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4.

mTORC1 and PKB/Akt control the muscle response to denervation by regulating autophagy and HDAC4.

Lysosomal degradation of newly formed insulin granules contributes to β cell failure in diabetes.

Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning.

Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning.

Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning.

Altered adipocyte differentiation and unbalanced autophagy in type 2 Familial Partial Lipodystrophy: an in vitro and in vivo study of adipose tissue browning.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Autophagy inhibition-mediated epithelial-mesenchymal transition augments local myofibroblast differentiation in pulmonary fibrosis.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Cytoplasmic DAXX drives SQSTM1/p62 phase condensation to activate Nrf2-mediated stress response.

Buffering of cytosolic calcium plays a neuroprotective role by preserving the autophagy-lysosome pathway during MPP+-induced neuronal death.

Buffering of cytosolic calcium plays a neuroprotective role by preserving the autophagy-lysosome pathway during MPP+-induced neuronal death.

Buffering of cytosolic calcium plays a neuroprotective role by preserving the autophagy-lysosome pathway during MPP+-induced neuronal death.

Buffering of cytosolic calcium plays a neuroprotective role by preserving the autophagy-lysosome pathway during MPP+-induced neuronal death.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

G3BP1 inhibits ubiquitinated protein aggregations induced by p62 and USP10.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

SQSTM-1/p62 potentiates HTLV-1 Tax-mediated NF-κB activation through its ubiquitin binding function.

Autophagy protein NRBF2 has reduced expression in Alzheimer's brains and modulates memory and amyloid-beta homeostasis in mice.

Autophagy protein NRBF2 has reduced expression in Alzheimer's brains and modulates memory and amyloid-beta homeostasis in mice.

Loss of autophagy impairs physiological steatosis by accumulation of NCoR1.

Loss of autophagy impairs physiological steatosis by accumulation of NCoR1.

Loss of autophagy impairs physiological steatosis by accumulation of NCoR1.

Autophagy Deficiency in Renal Proximal Tubular Cells Leads to an Increase in Cellular Injury and Apoptosis under Normal Fed Conditions.

Autophagy Deficiency in Renal Proximal Tubular Cells Leads to an Increase in Cellular Injury and Apoptosis under Normal Fed Conditions.

Autophagy Deficiency in Renal Proximal Tubular Cells Leads to an Increase in Cellular Injury and Apoptosis under Normal Fed Conditions.

AMPK-dependent activation of the Cyclin Y/CDK16 complex controls autophagy.

AMPK-dependent activation of the Cyclin Y/CDK16 complex controls autophagy.

AMPK-dependent activation of the Cyclin Y/CDK16 complex controls autophagy.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Microglia clear neuron-released α-synuclein via selective autophagy and prevent neurodegeneration.

Myocardial cathepsin D is downregulated in sudden cardiac death.

Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination.

Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination.

Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination.

Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination.

Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination.

Inhibition of striatonigral autophagy as a link between cannabinoid intoxication and impairment of motor coordination.

A Scaffold-Free 3-D Co-Culture Mimics the Major Features of the Reverse Warburg Effect In Vitro.

A Scaffold-Free 3-D Co-Culture Mimics the Major Features of the Reverse Warburg Effect In Vitro.

ISG15 Connects Autophagy and IFN-γ-Dependent Control of Toxoplasma gondii Infection in Human Cells.

ISG15 Connects Autophagy and IFN-γ-Dependent Control of Toxoplasma gondii Infection in Human Cells.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

Novel Insights into the Cellular Localization and Regulation of the Autophagosomal Proteins LC3A, LC3B and LC3C.

The Polyphenol Pterostilbene Ameliorates the Myopathic Phenotype of Collagen VI Deficient Mice via Autophagy Induction.

Degradation of connexin 50 protein causes waterclefts in human lens.

Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment.

Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment.

Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment.

Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment.

Prodigiosin Sensitizes Sensitive and Resistant Urothelial Carcinoma Cells to Cisplatin Treatment.

Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis.

Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis.

Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis.

Autophagy of the m6A mRNA demethylase FTO is impaired by low-level arsenic exposure to promote tumorigenesis.

N-Acetyl-L-leucine improves functional recovery and attenuates cortical cell death and neuroinflammation after traumatic brain injury in mice.

High-throughput screening for natural compound-based autophagy modulators reveals novel chemotherapeutic mode of action for arzanol.

High-throughput screening for natural compound-based autophagy modulators reveals novel chemotherapeutic mode of action for arzanol.

An essential role of the autophagy activating kinase ULK1 in snRNP biogenesis.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Phosphoproteomic identification of ULK substrates reveals VPS15-dependent ULK/VPS34 interplay in the regulation of autophagy.

Different Roles of p62 (SQSTM1) Isoforms in Keratin-Related Protein Aggregation.

Different Roles of p62 (SQSTM1) Isoforms in Keratin-Related Protein Aggregation.

Different Roles of p62 (SQSTM1) Isoforms in Keratin-Related Protein Aggregation.

FIP200 controls the TBK1 activation threshold at SQSTM1/p62-positive condensates.

Lack of Cathepsin D in the central nervous system results in microglia and astrocyte activation and the accumulation of proteinopathy-related proteins.

Malin restoration as proof of concept for gene therapy for Lafora disease.

BALB.NCT-Cpox nct is a unique mouse model of hereditary coproporphyria.